Non-neurogenic etiologies show a 1:1 HR response to βin BP (mmHg).
Consider:
π΄ Hypovolemia (hemorrhage, β intake, β losses)
π΄ Medications (alpha blockers, psychiatric meds)
Consider:
π΄ Hypovolemia (hemorrhage, β intake, β losses)
π΄ Medications (alpha blockers, psychiatric meds)
Patient history consistent with a specific etiology should prompt targeted testing.
BUT!
Patient descriptions are inconsistent & don't correlate well w/ underlying etiology; therefore, a more consistent Dx approach is to categorize dizziness as Acute vs. Chronic.
BUT!
Patient descriptions are inconsistent & don't correlate well w/ underlying etiology; therefore, a more consistent Dx approach is to categorize dizziness as Acute vs. Chronic.
I said I'd cover a tricky don't miss Dx: let's step into the world of acute, continuous dizziness.
DDx = posterior circulation stroke (!) vs. vestibular neuritis (benign)
If patient remains acutely dizzy, perform HINTS exam: youtube.com
DDx = posterior circulation stroke (!) vs. vestibular neuritis (benign)
If patient remains acutely dizzy, perform HINTS exam: youtube.com
More thorough Dx approach to dizziness by @AaronLBerkowitz here. Buy his fantastic book!
References:
1) Dr. John Chang (all-star clinician educator @YaleIM_Chiefs)
2) n.neurology.org
3) accessmedicine.mhmedical.com by @AaronLBerkowitz
4) youtube.com
1) Dr. John Chang (all-star clinician educator @YaleIM_Chiefs)
2) n.neurology.org
3) accessmedicine.mhmedical.com by @AaronLBerkowitz
4) youtube.com
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