18 Tweets 58 reads Jan 11, 2021
Based off of Run the List Episode 24, here is a #tweetorial on💘Tachyarrhythmias💘
🎧Full episode: bit.ly
Let's start with an EKG 👇 (@medicine_strong). For more practice, check out: bit.ly
Thanks to @Gurleen_Kaur96 for this week's edition!
❓What tachyarrhythmia is shown on that EKG above❓
Keep these things in mind when interpreting:
🔶Rate
🔶Regularity
🔶Width of QRS complex
🔶Relationship of P wave to QRS complex
And continue reading this 🧵 for the correct answer...
First, let's review some 🔑 principles:
🥇Always assess if patient is hemodynamically stable!
✅Hypotension
✅Altered Mental Status
✅Chest pain
✅Shortness of breath
If unstable ➡️ cardioversion or defibrillation
Now an aside on terminology:
💓Tachycardia💓 = HR >100, physiologic or pathologic
💘Tachyarrhythmia💘 = Heart rhythm disorder causing *pathologic* tachycardia
If sinus rhythm:
➡️P before each QRS wave
➡️QRS following each P
➡️P wave upright in lead II
Then ⏩ Sinus Tachycardia
🧠Let's develop a diagnostic framework for tachyarrhythmias
QRS complex on EKG is 💯
🔹Narrow complex (QRS <120ms): SVT
🔷Wide complex (QRS >120ms): SVT w/ aberrancy or ventricular tachycardia (V. tach)
Check out this ⤵️schema from @CPSolvers
SVT 🆚 V. tach
Quick tricks to identify V. tach on EKG:
✏️Concordance of precordial leads (all QRS complexes ➕ or all ➖)
✏️QRS complex ➖ in all inferior leads (II, III, aVF)
✏️AV dissociation w/ capture (QRS of normal duration) or fusion beats
Brugada Algorithm ⤵️
For *narrow* complex, next determine if irregular or regular
RTL schema by @haematognomist ⤵️
--
Ways to differentiate?
🪚Sawtooth pattern in inferior leads – A flutter
🌌Ectopic p wave – Atrial tachycardia
❎No p waves – A fib
🌈Irregularly regular, 3+ p waves – MAT
Now, back to the earlier EKG...
Answer❓ Multifocal Atrial Tachy (MAT)!
♦️Unknown MoA, triggered activity from delayed afterdepolarizations
Etiologies:
🫁COPD exacerbation, pneumonia, pulmonary embolism
💘CHF exacerbation
💊theophylline
♦️No one predominant P wave morphology!
🪂Now, let's take a deeper dive into AVNRT🪂
🔷Most common SVT
🔷More common in younger women
🔷Spontaneous events or by ☕️🤸🍵
🔷AV node re-entry circuit
90% Typical:
-slow AV pathway for anterograde
-fast path. for retrograde
10% Atypical:
-fast anterograde
-slow retrograde
For typical AVNRT:
Retrograde ⬅️ conduction typically occurs in the fast 🚀 pathway
Atria are activated either simultaneously with or just after the ventricles so P wave is in the ST segment or *buried* within the QRS complex
🔥So on EKG, the RP interval < PR interval🔥
We've covered a lot! Hopefully you're not😴
Let's hit it home with the treatment of SVT 🎯
Acute: Vagal stimulation (carotid sinus massage, Valsalva maneuver) or adenosine
💊Beta-blockers or calcium channel blockers can also suppress AVNRT event by blocking/slowing the AV node
⁉️ Modified Valsalva ⁉️
The REVERT study from 🇬🇧 randomized patients presenting w/ SVT to either modified or standard Valsalva
Conclusion:
-Modified Valsalva should be considered as first-line treatment!
Video of Modified Valsalva: bit.ly
Radiofrequency catheter ablation 🔪 is first-line therapy for symptomatic chronic AVNRT (palpitations, SOB, neck pulsations)
📢 Large registry studies report >95% success rates of slow-pathway ablation (preferred 🎯), with a <1% risk of AV block
🧊Cryoablation is alternative🧊
💥Summary/pearls for Tachyarrhythmias💥
1. Assess hemodynamic instability
2. EKG is 🗝️ - wide or narrow QRS
3. If narrow, determine if regular or irregular rhythm
Other labs/imaging: BMP, TSH, Troponin, BNP, urine toxicology, Echo
RTL Episode Handout: bit.ly

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