I believe there is too much focus on lactate (and LT1) as a training zone event i.e. stay below, as opposed to what it is, an indirect marker of cellular metabolism.
1/
are either
1) Misunderstanding the role of lactate
2/
3) Copying the Norwegians
4) Jumping on the latest fad
I'd even go further to say that continuous lactate monitoring will on confuse the picture, much like Glucose monitoring.
3/
I.e CGM for diabetics is a game changer, because they HAVE to focus on what to them is more than the minutiae (every carb counts as an insulin using diabetic)
And..
4/
But for the other 99.99% of us?
5/
Why? I am not going to go into detail here, but I will in another thread. But lactate is complicated.
Far from the waste fuel it once was. Lactate in the blood is not the exact measure you think it is.
You are looking at value which represents the result of:
6/
And what we now know (which even Brooks postulated a long time ago) lactate is the end point of glycolysis, not pyruvate.
This is a good but heavy read.
ncbi.nlm.nih.gov
7/
Then some appears in the blood, did I mention that cells all around the body can use lactate including the heart?
ncbi.nlm.nih.gov
8/
Far from the aerobic/anaerobic on/off switch it was once thought to represent.
And don't even get me started on 'lactic acid!'
"He/she is bathing in lactic acid" said the commentator.
9/
Nothing truly magical changes as you cross LT1, but this marks that point where lactate rises above baseline, and it IS an important metric.
But, I don't think it's one that should be used to train by, unless you are truly elite.
10/
Forget the lactate monitoring, although consider getting a properly done (the other issue with field lactate testing), lactate profile.
11/
No, you don't need to train a tonne just below LT1 (Zone 2) to improve it.
An improvement in LT1 (more power/speed) is just a function of increased aerobic fitness.
12/
Threshold, polarised, etc.
The key is proper training load management AND adequate recovery.
Not sticking to a specific lactate.
End.
13/
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